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π Hypercalcemia Main
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π¬ Hypercalcemia Diagnostic Algorithm
PTH-Based Systematic Approach to Differential Diagnosis
Systematic Approach to Hypercalcemia (Ca++ > 10.5 mg/dL)
STEP 1: CONFIRM HYPERCALCEMIA
Corrected Ca = Measured Ca +
0.8 Γ (4.0 - Albumin)
Or check ionized calcium
STEP 2: CHECK PTH LEVEL
Critical first branch point
(Normal range: 15-65 pg/mL)
PTH β₯20 pg/mL
(Elevated or Inappropriately Normal)
PTH <20 pg/mL
(Suppressed)
PTH-DEPENDENT HYPERCALCEMIA
Parathyroid-mediated causes
PRIMARY HPT (85%)
β’ Adenoma (80-85%)
β’ Hyperplasia (10-15%)
β’ Carcinoma (<1%)
Labs: PTHβ, Phosβ
1,25(OH)βD β/N
Next: Neck imaging
FHH (5%)
Familial Hypocalciuric
Hypercalcemia
Key: 24h urine Ca
Ca/Cr clearance
ratio <0.01
Family history +
OTHER (10%)
β’ Tertiary HPT
β’ Lithium-induced
β’ Thiazide unmasking
Consider med history
Check CKD status
PTH-INDEPENDENT HYPERCALCEMIA
Non-parathyroid causes
Ca++ >13 mg/dL?
High suspicion for malignancy
(80-90% probability)
MALIGNANCY WORKUP
1. Check PTHrP
2. SPEP/UPEP
3. CT chest/abd/pelvis
4. Check 1,25(OH)βD
PTHrP+ β Humoral
1,25β β Lymphoma
Lytic β Myeloma/Mets
VITAMIN D EVAL
Check:
β’ 25(OH)D level
β’ 1,25(OH)βD level
25ββ β Intoxication
1,25ββ + 25N/β β
Granulomas/Lymphoma
(Check ACE, imaging)
OTHER CAUSES
β’ Thyrotoxicosis
β’ Adrenal insufficiency
β’ Vitamin A toxicity
β’ Immobilization
β’ Milk-alkali syndrome
Check: TSH, cortisol,
medication history
π ADDITIONAL WORKUP FOR ALL PATIENTS
β’ Complete metabolic panel (Ca, Phos, Mg, Cr)
β’ 24-hour urine calcium and creatinine
β’ Alkaline phosphatase (bone turnover)
β’ Consider: ECG (short QT), renal ultrasound
β οΈ If Ca >14 mg/dL β EMERGENCY
Start treatment immediately while working up
π¬ KEY LABORATORY PATTERNS
DIAGNOSIS
PTH
PTHrP
25(OH)D
1,25(OH)βD
Phosphate
KEY FINDING
Primary HPT
ββ
Normal
Normal
β or N
β
Most common outpatient cause
Malignancy (PTHrP)
β
ββ
Normal
β or N
β
Squamous cell ca, RCC
Lymphoma
β
Normal
N/β
ββ
N/β
Extra-renal 1Ξ±-hydroxylase
Vitamin D intoxication
β
Normal
ββ
β
N/β
Check supplement history
Sarcoidosis
β
Normal
N/β
ββ
N/β
ACE β, hilar lymphadenopathy
π‘ CRITICAL CLINICAL PEARLS
π΄ PTH is the FIRST test - determines entire diagnostic pathway
π΄ "Inappropriately normal" PTH with hypercalcemia = primary hyperparathyroidism
π΄ Ca++ >13 mg/dL with suppressed PTH = malignancy until proven otherwise
π΄ Check 1,25(OH)βD if PTH suppressed - elevated suggests granulomas/lymphoma
β οΈ EMERGENCY if Ca >14 mg/dL or symptomatic - treat first, diagnose second!
π¨ TREATMENT PRIORITIES BY MECHANISM
PTH-MEDIATED:
β’ Surgery (parathyroidectomy)
β’ Cinacalcet if surgery not option
β’ Bisphosphonates for bones
NON-PTH MEDIATED:
β’ Treat underlying cause
β’ Steroids if granulomas/lymphoma
β’ Bisphosphonates + calcitonin
π MONITORING DURING WORKUP
β’ Serial calcium levels (daily if >12 mg/dL)
β’ Renal function (creatinine, BUN)
β’ ECG monitoring (QT interval, arrhythmias)
β’ Mental status assessment
Remember: PTH determines the pathway β Elevated/Normal PTH = Parathyroid problem | Suppressed PTH = Look elsewhere
Initial Evaluation
Critical Decision Point
PTH-Dependent Path
PTH-Independent Path
Additional Evaluation
Reference Information