Dysmorphic RBCs in Urinalysis

Recognizing glomerular hematuria: acanthocytes, thresholds, pitfalls, and when to escalate.

Phase-contrast microscopy Acanthocytes ≥20% Glomerular vs urologic

Why Dysmorphic RBCs Matter

  • Dysmorphic RBCs—especially acanthocytes—indicate glomerular bleeding.
  • They help distinguish GN from urologic sources (stone, tumor, infection).
  • They guide urgency: GN patterns warrant expedited workup and often nephrology consult.
Teaching Pearl: RBC casts are pathognomonic but uncommon; dysmorphic RBCs are more sensitive and practical to detect.

Definitions & Thresholds

  • Dysmorphic RBC: Misshapen erythrocyte from glomerular passage; spicules/blebs common.
  • Acanthocyte (G1 cell): Ring form with vesicle-like blebs—highly specific for glomerular origin.
  • Diagnostic Thresholds:
    • >5% dysmorphic RBCs suggests glomerular source.
    • >20% acanthocytes is highly specific for GN.
Data synthesized from nephrology urinalysis literature and microscopy standards.

Technique

  • Use fresh, midstream, clean-catch specimen (ideally examined within 2 hours).
  • Phase-contrast microscopy preferred; bright-field acceptable with training.
  • Scan low power for casts, then high power for morphology quantification.
If unavailable, partner with a lab using phase-contrast or send to a nephrology microscopy service.

Specificity, Sensitivity, and Pitfalls

  • Specificity: Acanthocytes are highly specific; dysmorphia alone less so.
  • Sensitivity: Improves with fresh samples and phase-contrast technique.
  • Pitfalls: Prolonged storage, high osmolality, and severe hematuria can distort RBCs.
Avoid: Over-calling dysmorphia in old specimens or after vigorous exercise.

When to Escalate

  • Dysmorphic RBCs or RBC casts + AKI/proteinuria → urgent nephrology consult.
  • Consider complements and serologies if GN suspected; expedite tissue diagnosis.
  • Gross hematuria with clots or terminal stream bleeding suggests urologic source—consider imaging/urology.
See also: GN Overview and RPGN emergency recognition.

Clinical Integration

  • Proteinuria + dysmorphic RBCs: Strengthens GN likelihood.
  • Hypertension/edema: Supports nephritic syndrome pattern.
  • Normal complements: Narrows differential (e.g., IgA nephropathy, ANCA, anti-GBM).
Case tie-in: See Case 22 (RPGN with IgA vasculitis) for real-world application.

References & Further Reading

  • KDIGO Glomerular Diseases guideline framework; microscopy best-practice reviews.
  • Studies validating acanthocyte thresholds for glomerular hematuria.
  • Educational atlases of urinary sediment morphology (phase-contrast).

For educational purposes only. Morphology thresholds require context and lab technique considerations.

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